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[DOI] [PMC free article] [PubMed] [Google Scholar] Cobine PA, Ojeda LD, Rigby KM, & Winge DR (2004)
Of note, pharmacologically induced PFC NMDAR hypofunction leads to increased oscillations [99] while genetic deletion of NMDAR1 subunits in pyramidal neurons increases oscillations, cell excitability and sEPSC frequency [100], similar to the present data
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H2S and its role in redox signaling
