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Conclusions These findings identify disrupted GSH homeostasis and inflammatory activation as metabolic features associated with symptomatic TTR-V30M amyloidosis in vivo, supporting a link between redox imbalance and disease manifestation
Gremese, E., Alivernini, S., Tolusso, B., Zeidler, M
C th l Glutathione gip ngn chn s hnh thnh ca cc sc t ny, lm gim s ti mu v mang n mt ln da trng sng mn mng, khng cn thm nm
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