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doi: 10.1007/s10787-024-01519-7 Summary Keywords atherosclerosis, endothelial dysfunction, ferroptosis, iron metabolism, lipid peroxidation, oxidized phospholipids, plaque instability Citation Li C, Wang J and Sun J (2026) Ferroptosis in arterial atherosclerosis: mechanistic hypotheses, cell type specific vulnerabilities, translational biomarkers, and therapeutic opportunities

Patients present with viral infections, atopic eczema, impaired T-cell function and TH17 cell differentiation, with defective eosinophil homeostasis and dysregulation of IgE (42, 43)

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Nam is present extracellularly and its uptake is possible either as direct transport in intact form or converted to salvage pathway metabolites
