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While it is said that any organ can be affected by mitochondrial defects, the brain, skeletal muscle, and myocardium are most often affected because of their higher aerobic activity and higher mitochondrial content [181]

The key role of histone acetylation in mGlu2 receptor regulation is also strengthened by the findings that chronic treatment with atypical antipsychotics down-regulates the expression of mGlu2 receptors in the mouse and human prefrontal cortex by increasing the binding of HDAC2 to the Grm2 promoter [39]

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