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Special attention is paid to the upregulation of hypoxia inducing factor-1 and the subsequent stimulation of the genes coding for glucose transporter-1 (GLUT-1) and the growth factors transforming growth factor- (TGF), vascular endothelial growth factor (VEGF), plasminogen growth factor activator inhibitor-1 (PAI-1) and connective tissue growth factor (CTGF)

Following injury to the central nervous system, mitochondrial dysfunction and generation of reactive oxygen species could lead to activation of neuronal and glial cell apoptosis [6], [42], [43]

What is sensed by ASJ and/or IL2, and why these upstream signals regulate, is unknown to us, and it will be very interesting to study them in the future

Reassess after 812 weeks