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These may include inhibitors of production/antagonists of the action of pro-inflammatory cytokines (e.g., infliximab) (Maini, 2004), compounds that decrease the production of adhesion molecules (e.g., -amyrin) (Shih and Cherng, 2014), antibodies targeting adhesion molecules (Siles et al., 2018), or compounds that compete with phagocytes to bind the endothelium (Pans et al., 1999)

Importantly, a microbiota-derived metabolite, taurodeoxycholic acid (TDCA), was elevated in CD progressors plasma, which proposed to have an enhancing effect on CD pathogenesis progression [64]

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