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While our recent studies demonstrated that the loss of AGXT exacerbates diet-induced MASH in mice 12 , the hepatic regulation of oxalate metabolism and its levels in humans and mice with MASH, the effects of oxalate accumulation in hepatocytes, the primary cells responsible for its formation and the therapeutic potential of targeting impaired oxalate metabolism in MASH, have not been systematically studied

Hypoxic-reoxygenated mice were fed normoxic for 28 days after receiving chronic hypoxia

doi: 10.1016/j.tox.2009.01.024 196 GeorgeASahinICarneiroBADizonDSSafranHPEl-DeiryWS

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