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While both scenarios likely contribute to immune tolerance, it is generally believed that hot tumors may respond better to immune checkpoint inhibitors (ICIs) than cold tumors because the former have pre-existing cytotoxic cells potentially capable of recognizing tumor derived neoantigens.47 For cold tumors, on the other hand, antitumor immunity is likely abrogated at a much earlier stepmaking this subtype potentially more challenging to treat with ICIs

however, the impact of an overgrowth of these bacteria and the effect of exposure to excessive amounts of bacteria-derived H 2 S are not known

* Thc phm ny khng phi l thuc, khng c tc dng thay th thuc cha bnh

61 Similarly, Uzi et al