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Endometrial receptivity vs

For example, when GlS1 is depleted, it promotes the differentiation and effector function of CD4 + Th1 and CD8 + T cells by up-regulating the expression of the transcription factor T-bet, and inhibiting the mTORC1 and IL-2 signal transduction pathways, thereby inhibiting Th17 differentiation [83]

doi: 10.1161/atvb.34.suppl_1.457
