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Although direct clinical evidence for anti-HCC activity in humans is lacking, its ability to attenuate insulin resistance, oxidative stress, and the inflammation-fibrosis cascade may indirectly reduce HCC risk by optimizing the hepatic oncogenic microenvironment

Escherichia coli, Proteus mirabilis, Acinetobacter lwoffii, Bacillus, Penicillium, and Rhizopus are among the additional microorganisms

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