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Description
Intriguingly, the interactions of N-terminal histidine residues of the prion protein (PrP) mainly with Cu 2+ sequestration and accumulation in diseased brain (T Frczyk, 2021) has long been identified to be responsible for the neurotoxic effects of PrP in neurons producing prion disease
The current G-LOC curves on the other hand provide a population standard for healthy human response to acceleration stress
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Supports hydration and combats dryness
