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Furthermore, Hcy accelerates endothelial cell (EC) aging by promoting apoptosis and other forms of cell death, including D-gasdermin-dependent pyroptosis [17]

After observing the cardiopulmonary tissue of a subset of patients with severe COVID-19 who also developed ARDS, immunothrombosis was identified as a significant contributing factor to these patients critical conditions [5,6]

This increase of necroptotic cells was associated with enhanced release of intracellular DAMPs as well as elevated NO and prostaglandin E2 production [124], which confirms the pro-inflammatory consequences of necroptosis

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